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Journal of Hepatology
Volume 51, Issue 3
, Pages 557-564
, September 2009
Hepatocyte GP73 expression in Wilson disease
References
- Disease- and cell-specific expression of GP73 in human liver disease. Am J Gastroenterol. 2004;99:1087
- GP73, a novel Golgi-localized protein upregulated by viral infection. Gene. 2000;249:53
- . Expression of GP73, a resident Golgi membrane protein, in viral and nonviral liver disease. Hepatology. 2002;35:1431
- . Endosomal trafficking and proprotein convertase cleavage of cis Golgi protein GP73 produces marker for hepatocellular carcinoma. Traffic. 2007;8:1415
- GP73, a resident Golgi glycoprotein, is a novel serum marker for hepatocellular carcinoma. J Hepatol. 2005;43:1007
- . Wilson’s disease. Clin Gastroenterol Hepatol. 2005;3:726
- Diagnosis and phenotypic classification of Wilson disease. Liver Int. 2003;23:139
- Diagnostic value of quantitative hepatic copper determination in patients with Wilson’s disease. Clin Gastroenterol Hepatol. 2005;3:811
- . Microwave energy for acid decomposition at elevated temperatures and pressures using biological and botanical samples. Anal Chem. 1986;58:2534
- . Chronic hepatitis. An update on terminology and reporting. Am J Surg Pathol. 1995;19:1409
- Null mutation of the murine ATP7B (Wilson disease) gene results in intracellular copper accumulation and late-onset hepatic nodular transformation. Hum Mol Genet. 1999;8:1665
- Consequences of copper accumulation in the livers of the Atp7b-/- (Wilson disease gene) knockout mice. Am J Pathol. 2006;168:423
- . Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) method. Methods. 2001;25:402
- . Relative expression software tool (REST) for group-wise comparison and statistical analysis of relative expression results in real-time PCR. Nucleic Acids Res. 2002;30:e36
- . Molecular pathogenesis of Wilson and Menkes disease: correlation of mutations with molecular defects and disease phenotypes. J Med Genet. 2007;44:673
- Genotype–phenotype correlations for a wide spectrum of mutations in the Wilson disease gene (ATP7B). Am J Med Genet A. 2004;131:168
- Wilson’s disease in patients presenting with liver disease: a diagnostic challenge. Gastroenterology. 1997;113:212
- . Structure, oxidant activity, and cardiovascular mechanisms of human ceruloplasmin. Life Sci. 1995;56:1749
- . A long-term follow-up study of the hepatic copper and serum ceruloplasmin concentrations in patients with chronic liver disease. Scand J Gastroenterol. 1982;17:545
- . Decreased survival and hepato-renal pathology in mice with C-terminally truncated GP73 (GOLPH2). Int J Clin Exp Pathol. 2009;2:34
- . Pathophysiology and clinical features of Wilson disease. Metab Brain Dis. 2004;19:229
- Relationship between oxidative stress and antioxidant systems in the liver of patients with Wilson disease: hepatic manifestation in Wilson disease as a consequence of augmented oxidative stress. Pediatr Res. 2006;60:472
- Oxidative stress and pro-apoptotic conditions in a rodent model of Wilson’s disease. Biochim Biophys Acta. 2005;1741:325
- Common human cancer genes discovered by integrated gene-expression analysis. PLoS ONE. 2007;2:e1149
- Golgi protein GOLM1 is a tissue and urine biomarker of prostate cancer. Neoplasia. 2008;10:1285
- Hepatocellular carcinoma associated with Wilson’s disease. Intern Med. 2004;43:1042
- . Hepatocellular carcinoma associated with an atypical presentation of Wilson’s disease. Semin Liver Dis. 2007;27:122
- High copper selectively alters lipid metabolism and cell cycle machinery in the mouse model of Wilson disease. J Biol Chem. 2007;282:8343
- . Cycling of early Golgi proteins via the cell surface and endosomes upon lumenal pH disruption. Traffic. 2002;3:641
- . Copper-dependent interaction of dynactin subunit p62 with the N-terminus of ATP7B but not ATP7A. J Biol Chem. 2006;281:14006
☆ This study was supported by an NIH grant (R21 DK075659) to S.L. The authors who have taken part in this study declared that they do not have anything to disclose regarding funding or conflict of interest with respect to this manuscript.
PII: S0168-8278(09)00398-5
doi: 10.1016/j.jhep.2009.05.029
© 2009 European Association for the Study of the Liver. Published by Elsevier Inc. All rights reserved.
« Previous
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Journal of Hepatology
Volume 51, Issue 3
, Pages 557-564
, September 2009
