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Letter to the Editor| Volume 68, ISSUE 4, P856-857, April 2018

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Energy drinks and adolescents – A hepatic health hazard?

Published:November 22, 2017DOI:https://doi.org/10.1016/j.jhep.2017.10.036
Energy drinks and adolescents – A hepatic health hazard?
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Next ArticleReply to: “Energy drinks and adolescents – A hepatic health hazard?”
      We read with great interest the paper entitled “Serum uric acid concentrations and fructose consumption are independently associated with NASH in children and adolescents”, published by Mosca et al. in the Journal of Hepatology, 2017.
      • Mosca A.
      • Nobili V.
      • De Vito R.
      • Crudele A.
      • Scorletti E.
      • Villani A.
      • et al.
      Serum uric acid concentrations and fructose consumption are independently associated with NASH in children and adolescents.
      J Hepatol. 2017; 66: 1031-1036

      Linked Article

      • Serum uric acid concentrations and fructose consumption are independently associated with NASH in children and adolescents
        Journal of HepatologyVol. 66Issue 5
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          Non-alcoholic fatty liver disease (NAFLD) is now largely regarded as the hepatic manifestation of metabolic syndrome (MetS) and NAFLD represents the most frequent chronic liver disease in children in Western countries [1]. NAFLD begins with the development of liver lipid accumulation and the condition progresses over time with the development of liver inflammation and fibrosis (non-alcoholic steatohepatitis or NASH). Although it was initially thought that NAFLD was a relatively harmless condition in children and adolescents, recent evidence shows that NASH occurs in this young population [2].
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      • Reply to: “Energy drinks and adolescents – A hepatic health hazard?”
        Journal of HepatologyVol. 68Issue 4
        • Preview
          We read with interest the letter by Robin et al. referring to our paper entitled “Serum uric acid concentrations and fructose consumption are independently associated with NASH in children and adolescents”.1,2 In their letter, the authors report the case of a 17-year-old-boy who developed NASH due to his unhealthy lifestyle and his consumption of energy drinks (EDs). Robin et al. also highlight that other components of the diet, such as niacin, xanthine, taurine and B vitamins, could contribute to liver damage.
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      References

        • Mosca A.
        • Nobili V.
        • De Vito R.
        • Crudele A.
        • Scorletti E.
        • Villani A.
        • et al.
        Serum uric acid concentrations and fructose consumption are independently associated with NASH in children and adolescents.
        J Hepatol. 2017; 66: 1031-1036
        View in Article

      2. NatCen Social Research, MRC Elsie Widdowson Laboratory UCLMS. National Diet and Nutrition Survey Years 1–6, 2008/09-2013/14. [data collection]. 8th Edition. UK Data Service. SN: 6533, doi: https://doi.org/10.5255/UKDA-SN-6533-7. 2017.

        View in Article
        • Mosca A.
        • Della Corte C.
        • Sartorelli M.R.
        • Ferretti F.
        • Nicita F.
        • Vania A.
        • et al.
        Beverage consumption and paediatric NAFLD.
        Eat Weight Disord. 2016; 21 (Available from:): 581-588
        http://link.springer.com/journal/40519
        View in Article
        • Breda J.J.
        • Whiting S.H.
        • Encarnação R.
        • Norberg S.
        • Jones R.
        • Reinap M.
        • et al.
        Energy drink consumption in Europe: a review of the risks, adverse health effects, and policy options to respond.
        Front Public Health. 2014; 2: 134
        View in Article
        • Mccrory C.
        • White C.M.
        • Bowman C.
        • Fenton N.
        • Reid J.L.
        • Hammond D.
        Perceptions and knowledge of caffeinated energy drinks : results of focus groups with Canadian youth.
        J Nutr Educ Behav. 2017; 49: 304-311
        View in Article
        • Zucconi S.
        • Volpato C.
        • Adinolfi F.
        • Gandini E.
        Gathering consumption data on specific consumer groups of energy drinks.
        EFSA J. 2013; (Available from): 1-190
        http://scholar.google.com/scholar?hl=en&btnG=Search&q=intitle:Gathering+consumption+data+on+specific+consumer+groups+of+energy+drinks#0
        View in Article

      7. Dietary Reference Values for Food Energy and Nutrients for the United Kingdom. London. HMSO 1991. [Report on Health and Social Subjects:41].

        View in Article
        • Harb J.N.
        • Taylor Z.A.
        • Khullar V.
        • Sattari M.
        Rare cause of acute hepatitis: A common energy drink.
        BMJ Case Rep. 2016; 2016 (Available from)
        http://casereports.bmj.com/
        View in Article
        • Vivekanandarajah A.
        • Ni S.
        • Waked A.
        Acute hepatitis in a woman following excessive ingestion of an energy drink: a case report.
        J Med Case Rep. 2011; 5: 227
        View in Article
        • Marventano S.
        • Salomone F.
        • Godos J.
        • Pluchinotta F.
        • Del Rio D.
        • Mistretta A.
        • et al.
        Coffee and tea consumption in relation with non-alcoholic fatty liver and metabolic syndrome: A systematic review and meta-analysis of observational studies.
        Clin Nutr. 2016; 35: 1269-1281
        View in Article

      Article info

      Publication history

      Published online: November 22, 2017

      Identification

      DOI: https://doi.org/10.1016/j.jhep.2017.10.036

      Copyright

      © 2017 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

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